Osteoarthritis protects against femoral neck fracture: The MEDOS study experience
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2017, BoneCitation Excerpt :It is unknown whether this is due to a subchondral bone defect or whether this results from the increased mechanical strain (load) on the joint. Serum levels of sclerostin are significantly reduced in OA patients [34], suggesting that the reduced osteocyte sclerostin contributes to systemic changes consistent with data that osteoarthritis is protective against hip fracture [35]. Because of the subchondral bone sclerosis, Radin, et al., have postulated that the increased stiffness contributes to the cartilage destruction [36].
Association of COL1A1 polymorphism with subchondral bone degeneration of the temporomandibular joint
2016, International Journal of Oral and Maxillofacial SurgeryCitation Excerpt :Therefore, genetic variations may elicit opposite effects in these two types of disease. This polymorphism negatively affects bone tissue in the aetiopathogenesis of osteoporosis18–20; thus, this polymorphism may be a protective factor against OA. However, in the present study, borderline significance was observed between the carriers of the GG homozygous genotype and other genotypes (GT/TT) in low-density lesions of TMJ OA (P = 0.043); the GG genotype may also be considered a risk factor for this type of TMJ OA (OR 1.643, 95% CI 1.016–2.658).
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