LetterAccepted Article
Open Access
Angiotensin-converting enzyme 2, a SARS-CoV-2 receptor, is upregulated by interleukin-6 via STAT3 signaling in synovial tissues
Sho Mokuda, Tadahiro Tokunaga, Junya Masumoto and Eiji Sugiyama
The Journal of Rheumatology July 2020, jrheum.200547; DOI: https://doi.org/10.3899/jrheum.200547
Sho Mokuda
From the Department of Clinical Immunology and Rheumatology, Hiroshima University Hospital, Hiroshima, Japan; Department of Pathology, Ehime University Proteo-Science Center and Graduate School of Medicine, Toon, Japan. This research was funded by JSPS KAKENHI (Grant Number 19K18499 to S.M., 19K08908 to E.S., 17H04656 and JP20H03719 to J.M.). Sho Mokuda, MD, PhD, Department of Clinical Immunology and Rheumatology, Hiroshima University Hospital, 1-2-3, Kasumi, Minami-ku, Hiroshima, 734-8551, Japan. E-mail: sho-mokuda@hiroshima-u.ac.jp
Tadahiro Tokunaga
From the Department of Clinical Immunology and Rheumatology, Hiroshima University Hospital, Hiroshima, Japan; Department of Pathology, Ehime University Proteo-Science Center and Graduate School of Medicine, Toon, Japan. This research was funded by JSPS KAKENHI (Grant Number 19K18499 to S.M., 19K08908 to E.S., 17H04656 and JP20H03719 to J.M.). Sho Mokuda, MD, PhD, Department of Clinical Immunology and Rheumatology, Hiroshima University Hospital, 1-2-3, Kasumi, Minami-ku, Hiroshima, 734-8551, Japan. E-mail: sho-mokuda@hiroshima-u.ac.jp
Junya Masumoto
From the Department of Clinical Immunology and Rheumatology, Hiroshima University Hospital, Hiroshima, Japan; Department of Pathology, Ehime University Proteo-Science Center and Graduate School of Medicine, Toon, Japan. This research was funded by JSPS KAKENHI (Grant Number 19K18499 to S.M., 19K08908 to E.S., 17H04656 and JP20H03719 to J.M.). Sho Mokuda, MD, PhD, Department of Clinical Immunology and Rheumatology, Hiroshima University Hospital, 1-2-3, Kasumi, Minami-ku, Hiroshima, 734-8551, Japan. E-mail: sho-mokuda@hiroshima-u.ac.jp
Eiji Sugiyama
From the Department of Clinical Immunology and Rheumatology, Hiroshima University Hospital, Hiroshima, Japan; Department of Pathology, Ehime University Proteo-Science Center and Graduate School of Medicine, Toon, Japan. This research was funded by JSPS KAKENHI (Grant Number 19K18499 to S.M., 19K08908 to E.S., 17H04656 and JP20H03719 to J.M.). Sho Mokuda, MD, PhD, Department of Clinical Immunology and Rheumatology, Hiroshima University Hospital, 1-2-3, Kasumi, Minami-ku, Hiroshima, 734-8551, Japan. E-mail: sho-mokuda@hiroshima-u.ac.jp
Abstract
The severe acute respiratory syndrome-coronavirus-2 (SARS-CoV-2) has spread explosively worldwide and resulted in a pandemic of a new respiratory disease called coronavirus disease 2019 (COVID-19) in 2020. Symptoms of COVID-19 include fever, malaise, cough, and in severe cases, pneumonia and acute respiratory distress syndrome (1,2).
In this issue
The Journal of Rheumatology
Vol. 51, Issue 4
1 Apr 2024
Accepted manuscript
Angiotensin-converting enzyme 2, a SARS-CoV-2 receptor, is upregulated by interleukin-6 via STAT3 signaling in synovial tissues
Sho Mokuda, Tadahiro Tokunaga, Junya Masumoto, Eiji Sugiyama
The Journal of Rheumatology Jul 2020, jrheum.200547; DOI: 10.3899/jrheum.200547