Abstract
Although we have drugs and biologics designed to interfere with the innate immune response, none has been shown to shut down the massive inflammatory reaction that occurs during sepsis. A new pro-inflammatory myeloid-cell receptor might make a better target.
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Nathan, C., Ding, A. TREM-1: A new regulator of innate immunity in sepsis syndrome. Nat Med 7, 530–532 (2001). https://doi.org/10.1038/87846
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DOI: https://doi.org/10.1038/87846
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