Special IssueFDG-PET Findings in the Wernicke-Korsakoff Syndrome
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The specificity of thalamic alterations in Korsakoff's syndrome: Implications for the study of amnesia
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2021, Current Research in NeurobiologyRelationship between white matter glucose metabolism and fractional anisotropy in healthy and schizophrenia subjects
2020, Psychiatry Research - NeuroimagingCitation Excerpt :Pathological changes in gray matter morphology, whether developmental (as in schizophrenia, bipolar and autism spectrum disorders) or neurodegenerative (as in various dementing conditions), leading to its expansion or loss, will necessarily engender maladaptive connectivity and heightened expenditure of energy in the white matter. Thus, increased glucose metabolism in the white matter has been shown in several pathological and experimental conditions of autoimmune, inflammatory, toxic, and traumatic etiology, such as with elevated intracranial pressure in idiopathic hydrocephalus syndrome (Ć gren-Wilsson et al., 2003) and experimental tissue compression (Floeth et al., 2013), in early systemic lupus erythematosus (Ramage et al., 2011), Wernicke-Korsakoff syndrome (Reed et al., 2003), experimental autoimmune encephalomyelitis (Radu et al., 2007; Buck et al., 2012), after cerebral irradiation (Esik et al., 1999, 2004) and carbon monoxide intoxication (Chou et al., 2019). Given the transient and reversible character of metabolic impairments in these conditions, they have been hypothesized to be due to dysmyelination, i.e. with implicit assumption of reductions in fractional anisotropy.
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