Research ArticleArticle
Monocytic Angiotensin and Endothelin Receptor Imbalance Modulate Secretion of the Profibrotic Chemokine Ligand 18
Judith Rademacher, Angela Kill, Kathrin Mattat, Duska Dragun, Elise Siegert, Jeannine Günther and Gabriela Riemekasten
The Journal of Rheumatology January 2016, jrheum.150474; DOI: https://doi.org/10.3899/jrheum.150474
Judith Rademacher
From the Department of Rheumatology and Clinical Immunology, and the Department of Nephrology, Transplantology and Intensive Care, University Hospital Charité; Cell Autoimmunity, German Rheumatism Research Center (DRFZ), a Leibniz Institute, Berlin; Department of Rheumatology, University of Lübeck, Lübeck, Germany. Supported by the intramural grant of the University Hospital Charité, by the German Systemic Sclerosis Network, the Scleroderma Foundation, the Mirjam Lichy Foundation, and the German Research Foundation grant RI 1056-11. J. Rademacher, MD, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ; A. Kill, MD, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ; K. Mattat, Department of Rheumatology and Clinical Immunology, Charité University Hospital; D. Dragun, Professor, Department of Nephrology, Transplantology and Intensive Care, University Hospital Charité; E. Siegert, MD, Department of Rheumatology and Clinical Immunology, Charité University Hospital; J. Günther*, MSc, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ; G. Riemekasten*, Professor, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ, and Department of Rheumatology, University of Lübeck. * These authors are equivalent senior authors. Address correspondence to Dr. G. Riemekasten, Department of Rheumatology, University of Lübeck, Ratzeburger Allee 160, 23538 Lübeck, Germany. E-mail: Gabriela.Riemekasten@uksh.de. Accepted for publication November 2, 2015.
Angela Kill
From the Department of Rheumatology and Clinical Immunology, and the Department of Nephrology, Transplantology and Intensive Care, University Hospital Charité; Cell Autoimmunity, German Rheumatism Research Center (DRFZ), a Leibniz Institute, Berlin; Department of Rheumatology, University of Lübeck, Lübeck, Germany. Supported by the intramural grant of the University Hospital Charité, by the German Systemic Sclerosis Network, the Scleroderma Foundation, the Mirjam Lichy Foundation, and the German Research Foundation grant RI 1056-11. J. Rademacher, MD, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ; A. Kill, MD, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ; K. Mattat, Department of Rheumatology and Clinical Immunology, Charité University Hospital; D. Dragun, Professor, Department of Nephrology, Transplantology and Intensive Care, University Hospital Charité; E. Siegert, MD, Department of Rheumatology and Clinical Immunology, Charité University Hospital; J. Günther*, MSc, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ; G. Riemekasten*, Professor, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ, and Department of Rheumatology, University of Lübeck. * These authors are equivalent senior authors. Address correspondence to Dr. G. Riemekasten, Department of Rheumatology, University of Lübeck, Ratzeburger Allee 160, 23538 Lübeck, Germany. E-mail: Gabriela.Riemekasten@uksh.de. Accepted for publication November 2, 2015.
Kathrin Mattat
From the Department of Rheumatology and Clinical Immunology, and the Department of Nephrology, Transplantology and Intensive Care, University Hospital Charité; Cell Autoimmunity, German Rheumatism Research Center (DRFZ), a Leibniz Institute, Berlin; Department of Rheumatology, University of Lübeck, Lübeck, Germany. Supported by the intramural grant of the University Hospital Charité, by the German Systemic Sclerosis Network, the Scleroderma Foundation, the Mirjam Lichy Foundation, and the German Research Foundation grant RI 1056-11. J. Rademacher, MD, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ; A. Kill, MD, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ; K. Mattat, Department of Rheumatology and Clinical Immunology, Charité University Hospital; D. Dragun, Professor, Department of Nephrology, Transplantology and Intensive Care, University Hospital Charité; E. Siegert, MD, Department of Rheumatology and Clinical Immunology, Charité University Hospital; J. Günther*, MSc, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ; G. Riemekasten*, Professor, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ, and Department of Rheumatology, University of Lübeck. * These authors are equivalent senior authors. Address correspondence to Dr. G. Riemekasten, Department of Rheumatology, University of Lübeck, Ratzeburger Allee 160, 23538 Lübeck, Germany. E-mail: Gabriela.Riemekasten@uksh.de. Accepted for publication November 2, 2015.
Duska Dragun
From the Department of Rheumatology and Clinical Immunology, and the Department of Nephrology, Transplantology and Intensive Care, University Hospital Charité; Cell Autoimmunity, German Rheumatism Research Center (DRFZ), a Leibniz Institute, Berlin; Department of Rheumatology, University of Lübeck, Lübeck, Germany. Supported by the intramural grant of the University Hospital Charité, by the German Systemic Sclerosis Network, the Scleroderma Foundation, the Mirjam Lichy Foundation, and the German Research Foundation grant RI 1056-11. J. Rademacher, MD, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ; A. Kill, MD, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ; K. Mattat, Department of Rheumatology and Clinical Immunology, Charité University Hospital; D. Dragun, Professor, Department of Nephrology, Transplantology and Intensive Care, University Hospital Charité; E. Siegert, MD, Department of Rheumatology and Clinical Immunology, Charité University Hospital; J. Günther*, MSc, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ; G. Riemekasten*, Professor, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ, and Department of Rheumatology, University of Lübeck. * These authors are equivalent senior authors. Address correspondence to Dr. G. Riemekasten, Department of Rheumatology, University of Lübeck, Ratzeburger Allee 160, 23538 Lübeck, Germany. E-mail: Gabriela.Riemekasten@uksh.de. Accepted for publication November 2, 2015.
Elise Siegert
From the Department of Rheumatology and Clinical Immunology, and the Department of Nephrology, Transplantology and Intensive Care, University Hospital Charité; Cell Autoimmunity, German Rheumatism Research Center (DRFZ), a Leibniz Institute, Berlin; Department of Rheumatology, University of Lübeck, Lübeck, Germany. Supported by the intramural grant of the University Hospital Charité, by the German Systemic Sclerosis Network, the Scleroderma Foundation, the Mirjam Lichy Foundation, and the German Research Foundation grant RI 1056-11. J. Rademacher, MD, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ; A. Kill, MD, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ; K. Mattat, Department of Rheumatology and Clinical Immunology, Charité University Hospital; D. Dragun, Professor, Department of Nephrology, Transplantology and Intensive Care, University Hospital Charité; E. Siegert, MD, Department of Rheumatology and Clinical Immunology, Charité University Hospital; J. Günther*, MSc, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ; G. Riemekasten*, Professor, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ, and Department of Rheumatology, University of Lübeck. * These authors are equivalent senior authors. Address correspondence to Dr. G. Riemekasten, Department of Rheumatology, University of Lübeck, Ratzeburger Allee 160, 23538 Lübeck, Germany. E-mail: Gabriela.Riemekasten@uksh.de. Accepted for publication November 2, 2015.
Jeannine Günther
From the Department of Rheumatology and Clinical Immunology, and the Department of Nephrology, Transplantology and Intensive Care, University Hospital Charité; Cell Autoimmunity, German Rheumatism Research Center (DRFZ), a Leibniz Institute, Berlin; Department of Rheumatology, University of Lübeck, Lübeck, Germany. Supported by the intramural grant of the University Hospital Charité, by the German Systemic Sclerosis Network, the Scleroderma Foundation, the Mirjam Lichy Foundation, and the German Research Foundation grant RI 1056-11. J. Rademacher, MD, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ; A. Kill, MD, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ; K. Mattat, Department of Rheumatology and Clinical Immunology, Charité University Hospital; D. Dragun, Professor, Department of Nephrology, Transplantology and Intensive Care, University Hospital Charité; E. Siegert, MD, Department of Rheumatology and Clinical Immunology, Charité University Hospital; J. Günther*, MSc, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ; G. Riemekasten*, Professor, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ, and Department of Rheumatology, University of Lübeck. * These authors are equivalent senior authors. Address correspondence to Dr. G. Riemekasten, Department of Rheumatology, University of Lübeck, Ratzeburger Allee 160, 23538 Lübeck, Germany. E-mail: Gabriela.Riemekasten@uksh.de. Accepted for publication November 2, 2015.
Gabriela Riemekasten
From the Department of Rheumatology and Clinical Immunology, and the Department of Nephrology, Transplantology and Intensive Care, University Hospital Charité; Cell Autoimmunity, German Rheumatism Research Center (DRFZ), a Leibniz Institute, Berlin; Department of Rheumatology, University of Lübeck, Lübeck, Germany. Supported by the intramural grant of the University Hospital Charité, by the German Systemic Sclerosis Network, the Scleroderma Foundation, the Mirjam Lichy Foundation, and the German Research Foundation grant RI 1056-11. J. Rademacher, MD, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ; A. Kill, MD, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ; K. Mattat, Department of Rheumatology and Clinical Immunology, Charité University Hospital; D. Dragun, Professor, Department of Nephrology, Transplantology and Intensive Care, University Hospital Charité; E. Siegert, MD, Department of Rheumatology and Clinical Immunology, Charité University Hospital; J. Günther*, MSc, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ; G. Riemekasten*, Professor, Department of Rheumatology and Clinical Immunology, Charité University Hospital, and Cell Autoimmunity, DRFZ, and Department of Rheumatology, University of Lübeck. * These authors are equivalent senior authors. Address correspondence to Dr. G. Riemekasten, Department of Rheumatology, University of Lübeck, Ratzeburger Allee 160, 23538 Lübeck, Germany. E-mail: Gabriela.Riemekasten@uksh.de. Accepted for publication November 2, 2015.
In this issue
The Journal of Rheumatology
Vol. 51, Issue 4
1 Apr 2024
Monocytic Angiotensin and Endothelin Receptor Imbalance Modulate Secretion of the Profibrotic Chemokine Ligand 18
Judith Rademacher, Angela Kill, Kathrin Mattat, Duska Dragun, Elise Siegert, Jeannine Günther, Gabriela Riemekasten
The Journal of Rheumatology Jan 2016, jrheum.150474; DOI: 10.3899/jrheum.150474
Monocytic Angiotensin and Endothelin Receptor Imbalance Modulate Secretion of the Profibrotic Chemokine Ligand 18
Judith Rademacher, Angela Kill, Kathrin Mattat, Duska Dragun, Elise Siegert, Jeannine Günther, Gabriela Riemekasten
The Journal of Rheumatology Jan 2016, jrheum.150474; DOI: 10.3899/jrheum.150474