EditorialEditorial
Diabetogenesis in Rheumatoid Arthritis: Do Inflammation and Glucocorticoid-induced Incretin Overproduction Cause β-Cell Overcompensation and Consequent Premature Decompensation?
PATRICK H. DESSEIN, ANNE E. STANWIX and AHMED SOLOMON
The Journal of Rheumatology March 2019, 46 (3) 219-222; DOI: https://doi.org/10.3899/jrheum.181163
PATRICK H. DESSEIN
Cardiovascular Pathophysiology, and Genomics Research Unit, School of Physiology, Faculty of Health Sciences, University of the Witwatersrand, and Department of Rheumatology, Charlotte Maxeke Johannesburg Academic Hospital, Faculty of Health Sciences, University of the Witwatersrand, and Free University and University Hospital, Brussels,
Belgium;
ANNE E. STANWIX
Department of Rheumatology, Charlotte Maxeke Johannesburg Academic Hospital, Faculty of Health Sciences, University of the Witwatersrand;
AHMED SOLOMON
Department of Rheumatology, Charlotte Maxeke Johannesburg Academic Hospital, Faculty of Health Sciences, University of the Witwatersrand, Johannesburg,
South Africa.

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In this issue
The Journal of Rheumatology
Vol. 46, Issue 3
1 Mar 2019
Diabetogenesis in Rheumatoid Arthritis: Do Inflammation and Glucocorticoid-induced Incretin Overproduction Cause β-Cell Overcompensation and Consequent Premature Decompensation?
PATRICK H. DESSEIN, ANNE E. STANWIX, AHMED SOLOMON
The Journal of Rheumatology Mar 2019, 46 (3) 219-222; DOI: 10.3899/jrheum.181163
Diabetogenesis in Rheumatoid Arthritis: Do Inflammation and Glucocorticoid-induced Incretin Overproduction Cause β-Cell Overcompensation and Consequent Premature Decompensation?
PATRICK H. DESSEIN, ANNE E. STANWIX, AHMED SOLOMON
The Journal of Rheumatology Mar 2019, 46 (3) 219-222; DOI: 10.3899/jrheum.181163